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Deletion of the SOAR/CAD domain abolishes store depletion-induced clustering of Orai1 monomers and dramatically reduces SOCE, suggesting this domain is required for the assembly of the SOCE complex at ER-PM junctions (76, 77)
The ME/CFS research field has been stuck in a somewhat defensive posture, with a focus on demonstrating this is a real condition by showing significant biological differences between patients and controls
It reduces melanin production by inhibiting the enzyme tyrosinase, which is responsible for pigment formation
Cyclic AMP, like uric acid, is a known endogenous substrate for MRP4
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Germoush et al
