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The observations that GLP-1R signaling in cells is essential for control of insulin secretion, together with data suggesting that exendin(9-39) may attenuate insulin secretion through reduction of cell cyclic AMP (97), has supported investigational use of exendin(9-39) for the treatment of severe hyperinsulinemic hypoglycemia

First and foremost, GLP-1 mediated signaling in the brain has a well-established neuroprotective effect, which has been repeatedly proven to enhance memory and cognitive functions, improve neurogenesis and synaptic functions, as well as restore balance in neurotransmission, i.e

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