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Muscle injuries induce a prostacyclin-PPARgamma/PGC1a-FAO spike that boosts regeneration

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The tubular protective effects appeared to be associated with the counteraction of PPAR deterioration, resulting in a maintenance of FAO, a decrease of intracellular accumulation of undigested FFAs, and attenuation of disease developmental factors including oxidative stress, apoptosis, and NFB activation (Figure 2)

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Together, these three sub-sections comprehensively evaluate how L-carnitine supplementation affects performance during high-intensity exercise

Compound C was preincubated with FLS for 12 h to inhibit the activation of AMPK in advance, and FLS were then treated with l-carnitine for another 24 h