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The novel mechanism by which 4-HNE inhibits NLRP3 is through its ability to directly bind to the critical domains of NLRP3, disrupting its interaction with the adapter protein NEK7, a key step in inflammasome activation, thereby preventing caspase-1 activation

It reinforces the understanding that thalassemia is not limited to blood counts, but involves deeper cellular-level dysfunction

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Technological Highlights: Micro-Beams: Concentrates the energy on small points for faster healing