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Reprod BioMed Online (2007) 14(6):72733

This targeted approach to therapy allows for precise modulation of biological functions, leading to favorable outcomes for patients

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From these results, it could be inferred that CaOx-induced ferroptosis, as observed in the previous series of biochemical reactions, reduced the ability of cells to resist oxidation, thereby resulting in Fe2+ overload, mitochondrial damage and an increase in lipid peroxidation, which then induced an intracellular ROS outbreak, eventually causing severe cell injury

[DOI] [PMC free article] [PubMed] [Google Scholar] 18.imrov B., Kraus V., Jr., Birkov A

Hepatogastroenterology 2009;56:1719-1723