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In human fetal pulmonary microvascular endothelial cells (HPMEC) with AhR deficit, a decrease in AhR expression, an increase in reactive oxygen species (ROS) formation, and a weakening of SOD1 activity were observed [86]

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Apoptosis-inducing factor, mitochondrion-associated 2, regulates Klf1 in a mouse erythroleukemia cell line

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