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CONCLUSIONS The literature suggests that the pathophysiology of DE/AE is multifactorial, including both organic and psychosocial factors that are neither independent nor mutually exclusive

Abnormally elevated levels of DRP1 not only cause mitochondrial fission, mitochondrial fragmentation, and decreased efficiency of mitochondrial ATP production but also impede glucose uptake (104) and glucose-stimulated insulin secretion by inhibiting the translocation of the insulin receptor substrate 1-protein kinase B (IRS1-Akt) pathway and glucose transport proteins in mouse pancreatic -cells (105)

Mol Biol Cell (2023) 34(9):ar91

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PMID: 32542159 Sion Scott, Helen May, Martyn Patel, David J Wright, Debi Bhattacharya