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Although the exact mechanistic role of EBV in cHL pathogenesis is unclear, the presence of EBV throughout disease progression underscores its role in maintaining the tumor phenotype (196)

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From what I've read & what I've read on here etc, as everyone is different, it does vary

1, this period can vary by animal and depends on the level of maternal antibody and the vaccine antigen, which presents a major challenge for vaccine development

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All of the mutations in IDH1 that are associated with gliomagenesis are missense mutations that alter the normal Arg residue at position 132 to a histidine (R132H)