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In TMZ-resistant GBM cells (such as A172-R and T98G-R cells), boric acid treatment significantly increased the expression level of NCOA4, elevated intracellular iron levels, and triggered lipid peroxidation and GSH depletion, thereby promoting ferritin autophagy and inducing ferroptosis (151)

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The 16-hydroxy and 4-hydroxy pathways are considered bad as they are linked to higher risks of breast cancer

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Katrina is currently using a mouse model and single cell RNA sequencing analysis approaches to investigate interactions between resident glial populations in the brain and metastatic breast cancer cells
