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An interesting example of an extension to Reeds GSH metabolism model has been presented by Kelly et al., [169] with the addition of quinone redox metabolism to the GSH model

No clinical trials have been completed in humans to date [8]

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On the other hand, in a human study, four-week chronic OXT administration stimulated the oxytocinergic system in children with ASD, leading to heightened OXT levels and reduced methylated sites in the OXTR [89]

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