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Since IGF-1R inhibition results in suppression of downstream AKT activation, the IGF-1R inhibitors (BMS-754807 and GSK1838705A) efficiently inhibit AKT phosphorylation, indicating that the drugs function properly

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While our data confirm that elevated levels of baseline cell death are a characteristic of ADA2-deficient cells, the phenotype is unresponsive to inhibitors of the necroptotic pathway

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