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In: Mariotti F, ed
The research showed that patients experienced meaningful improvements in both pain levels and functional ability
It binds and activates GHRH receptors on pituitary somatotrophs, directly stimulating growth hormone synthesis and pulsatile release the same signaling axis as endogenous GHRH, but massively extended
This makes thymosin beta-4 particularly relevant to fracture healing because it directly supports the cellular mobility required at every stage of bone repair
