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The present results indicated that the antihypertensive effect of 1 AR-selective antagonists may be explained by their ability to hamper norepinephrine release through presynaptic modulation, in agreement with previous analyses showing that the hypotensive effect of AR antagonists, including atenolol, did not depend on cardio depression or suppression of renin secretion (31)

Gowin JL, Sloan ME, Stangl BL, Vatsalya V, Ramchandani VA

N-acetylcysteine in the treatment of human arsenic poisoning

Reactive sulfur species: kinetics and mechanism of the oxidation of cystine by hypochlorous acid to give N,N-dichlorocystine
doi: 10.1111/scd.12459

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