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Sirtuins use the ADP-ribose produced upon NAD + cleavage as an acetyl acceptor to generate acetyl-ADP-ribose with NAM release

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Throughout the study, ROS concentration, as well as SOD1 and CAT activities, were monitored for a year, but no significant changes were observed

Extrapolated studies revealed that AST was able to protect neuroblastoma cells from amyloid toxicity by upregulating the HO-1 anti-oxidative enzyme of the haem pathway [95]