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Outcomes from other smaller trials (reviewed in 89) have suggested either a lack of an effect or the possibility of an increased risk of cancer

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While these agents initially block growth signals to induce cytostatic effects, therapy-tolerant DTPs and dying therapy-sensitive cells dynamically secrete signaling molecules to reprogram neighboring tumor behavior (Fig

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However, as DRAK2-deficient T cells bear enhanced CRAC activity (136), and DRAK2-transgenic thymocytes have diminished Ca 2+ mobilization following TCR stimulation (132), it is likely that DRAK2 may phosphorylate an intermediate in CRAC signaling

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