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Description
Brain banking for research into neurodegenerative disorders and ageing
In situation of an increase in hepatocellular cytosolic Cu, ATP7B localizes to an acidified vesicular compartment near the canalicular membrane to accumulate Cu 2+ for subsequent excretion into bile ( Figure 1 ) (27-30)
The observed inhibition of GSSG reduction likely leads to redox stress resulting in the potent schistosomicidal activity observed
The active group in its molecular structure is the sulfhydryl group (-SH) on the cysteine residue
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