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Recent research has identified and dubbed a novel regulatory cell death modalitycuprotosiswherein copper ions bind to acylated proteins in the tricarboxylic acid cycle of mitochondrial respiration, resulting in protein aggregation, subsequent downregulation of iron-sulfur cluster protein expression, induction of proteotoxic stress, and eventual cell death

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These stress sensors display a mode of activation similar to both each other and that of HSF1 in the HSR

The findings from this meta-analysis were significant and revealing

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