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The APE1 inefficiently cleaves but recruits various transcription factors to activate downstream genes, such as vascular endothelial growth factor (VEGF) and endonuclease IIIlike protein 1 (NTHL1) [95]

In macrophages, both HO-1 and its isoform HO-2 participate in the degradation of heme derived from senescent or damaged red blood cells, thereby playing a crucial role in maintaining systemic heme homeostasis through their enzymatic activities (46)

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