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Description
Notably, the BTK inhibitor and clinical compound ibrutinib showed additive effects with GPX4 inhibition, even at concentrations below its cytotoxic threshold, expanding its therapeutic relevance beyond BTK inhibition
Constant, discriminative, and almost non-overlapping relative shifts in mean TWCCSN2, meas were obtained for demethylation (6.52.1 2), oxygenation (hydroxylation+3.81.4 2, N-oxidation+3.43.3 2), acetylation (+13.51.9 2), sulfation (+17.94.4 2), glucuronidation (N-linked:+41.77.5 2, O-linked:+38.18.9 2), and glutathione conjugation (+49.213.2 2)
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