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265 Hence, understanding metabolic complexity not only provides insight into the biological inhibition of CSCs but also identifies actionable vulnerabilities that may be exploited to increase therapeutic efficacy

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Dong Yao, [email protected] These authors have contributed equally to this work and share first authorship TThese authors have contributed equally to this work and share last authorship Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

A large portion of the brain damage from TBIs comes from secondary damage when neighboring brain cells are killed by excessive extracellular glutamate and calcium ions, released by the physically damaged cells, which disrupts the homeostatic balance of intracellular calcium in bystander cells resulting in apoptosis