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Functionally, this amplification leads to overexpression of the c-Met protein, which brings about worse prognosis of the disease

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The subsequent upregulation of ferritin is essential to obtain the protective effect, as it can store the released iron in a catalitically inactive form (Balla et al., 2010)

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Serum levels of 4-hydroxynonenal adducts and responding autoantibodies correlate with the pathogenesis from hyperglycemia to Alzheimer's disease

BPC-157 significantly enhances ERK1/2 phosphorylation in a dose-dependent manner, activating downstream targets including transcription factors c-Fos, c-Jun, and Egr-1