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Kawashima, I
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When barrier integrity is impaired, microbial products may activate Toll-like receptor 4 (TLR4)-myeloid differentiation primary response gene 88 (MyD88)-nuclear factor-kappa B (NF-B) signaling and amplify proinflammatory cascades, further aggravating barrier dysfunction [67]

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Furthermore, GSH has been shown to react with newly-created free radicals, thereby protecting NO from oxidative damage and potentiating the effectiveness of NO [6]

doi:10.1002/jcp.22571, PMID: 21660956 Guha P, Dey A, Sen R, Chatterjee M, Chattopadhyay S, Bandyopadhyay SK (2011) Intracellular GSH depletion triggered mitochondrial Bax translocation to accomplish resveratrol-induced apoptosis in the U937 cell line
