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Because inositol-derived signalling regulates intracellular Ca release and Ca elevations can negatively modulate AKT activation, the preservation of Thr308 phosphorylation is particularly informative and supports the idea that Myo-Ins does not impair upstream AKT recruitment and activation, but instead selectively targets Ser473

Oxidative stress In -thalassemia, ineffective erythropoiesis results in increased hemolysis and the release of free hemoglobin, which further exacerbates oxidative stress through the Fenton reaction, where iron catalyzes the conversion of hydrogen peroxide into highly reactive hydroxyl radicals (76)

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