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The EBV latency III induces resistance to ferroptosis inducing agents To gain insights into how EBV latency programs alter lipid ROS levels and ferroptosis vulnerability, we utilized isogenic Burkitt tumor derived MUTU I vs III cell lines, which differ only by EBV latency I vs III programs, (43)

[DOI] [PubMed] [Google Scholar] Chen L, Patel RP, Teng X, Bosworth CA, Lancaster JR, Jr, Matalon S

Identification and validation of immune and cuproptosisRelated genes for diabetic nephropathy by WGCNA and machine learning

Subsequent pore formation following binding to cardiolipin may then facilitate entry of the oligomers into the intermembrane space, where they then gain access to much more cardiolipin present on the inner membrane

Body Composition Aging is associated with increased abdominal obesity and waist circumference (6)

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