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While sodium's significance in the context of epilepsy is evident, the specific mechanisms through which alterations in sodium channels or sodium levels contribute to epileptogenesis may differ based on the subtype or cause of epilepsy[147]

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Kerrigan, C

Clinical studies suggested the inflammatory activities in atherosclerotic plaques measured by 18F-fluorodeoxyglucose (FDG) PET or microcalcification tracked by 18F-sodium fluoride (NaF) PET combined with CT in atherosclerotic plaques are related to cardiovascular events [214,215,216]

Chromatin remodeling factors also play a pivotal role

a second VOI (40 12 18mm 3 ) was placed in the left insula
