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Metabolites derived from the diet can interact with the intestinal epithelium causing stimuli that may directly induce structural damage in epithelial cells and activate pro-carcinogenic signalling pathways, or by indirectly interacting with the gut microbiota (14)

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It is critical to reduce participant burden and guarantee that the study does not have a detrimental influence on their quality of life

Insulin resistance promotes ectopic lipid accumulation and systemic metabolic dysfunction

Similarly, patients interested in the metabolic and body-composition aspects of aging often explore how peptide protocols interact with weight-management strategies

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