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The reliance on OXPHOS makes CSCs resistant to conventional therapies targeting glycolysis-dependent cells, while their ability to enter quiescence and undergo EMT enhances their resilience and metastatic potential [90, 91]
10.1167/tvst.3.3.4 32 LeeS.IvesA
These toxins increase the demand for glutathione, as the liver uses it to neutralize and escort these substances out of the body
As an ectoenzyme, CD38 catalyzes conversion of NAD + to nicotinamide, which is then almost completely reused for NAD + synthesis via the salvage pathway (Fig
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On the other hand, in some cases of SIBO, bacteria from the large intestines travel back into the small intestines
