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Cytokine profiling revealed that only HA + GSH-C4 elicited significant modulation of tear mediators, and post-treatment cytokine levels correlated directly with other clinical metrics, consistent with a glutathione-mediated rebalancing of the ocular surface microenvironment

[17] Knockout studies with GPX1, GPX2, or GPX3 deficient mice showed that cytosolic GPX4 is so far the only glutathione peroxidase that is indispensable for embryonic development and cell survival

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doi: 10.3389/fpls.2018.01612

Nonetheless, the declining values in normal aging people are not considered to be diagnostic criteria for GHD or GH insufficiency worthy of treatment with rhGH