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Available at Takagi K, Suzuki F, Barrow RE, Wolf SE, Kobayashi M, Herndon DN

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Neurotoxicity, oxidative stress responses such as changes of GPX and GST activities, and metabolic dysfunction indicators such as ATPase activities may be the key endpoints of neonicotinoid toxicity as revealed from our study

Different flavonoid subclasses exert distinct effects on cancerrelated metabolic pathways

Due to bone thickening and fibrous dysplasia surgery is not usually an option for treatment and radiotherapy is contra-indicated because of the potential for sarcomatous change in fibrous dysplasia
