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Nejati et al., 2018), disrupting mitochondrial function, inducing apoptosis and autophagy, and inhibiting T and B cell proliferation ( H

AML with germline DDX41 variants is a clinicopathologically distinct entity with an indolent clinical course and favorable outcome

Varney said

Upregulation of BCL-2 by acridone derivative through gene promoter i-motif for alleviating liver damage of NAFLD/NASH

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