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Elevated intracellular GSH levels blocked bortezomib-induced nuclear factor erythroid 2-related factor 2 (NFE2L2, NRF2)-associated stress responses, including upregulation of the xCT subunit of the Xc- cystine-glutamate antiporter

FAT/CD36 functions both as an LCFA transporter and as a signaling receptor

As a pharmacologic therapeutic, optimizing the efficacy of GSH and minimizing its unintended effects means it is crucial to determine the correct timing, dose, and disease context (Schieber and Chandel, 2014)

During the 12 years that followed, he did research, dissection, and illustration for his highly acclaimed laboratory manuals on mammalian anatomy

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In: Hanif M.A., Nawaz H., Khan M.M., Byrne H.J., editors