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Glutamatergic System Counteracts NMDA Dysfunction in Schizophrenia Models BPC-157s glutamate modulation carries particular promise for treatment-resistant schizophrenia and NMDA receptor hypofunction disorders

5A, Supplementary Fig

Left untreated, a severe deficiency can cause permanent nerve damage

Stability of the arginine variant The BPC-157 stable salts patent described that salts containing two moles of basic amino acid per mole of pentadecapeptide, and especially the L-arginine salt, had the most favorable properties

The proteins examined are: HSL, ATGL, adiponutrin, triacylglycerol hydrolase, / hydrolase containing mRNA 5 (also known as CGI-58) and 11 (also known as Beuren-Williams Syndrome Critical Region 21), acylpeptide hydrolase, esterase 1, esterase D, carboxylesterase ML1, and membrane-associated calcium-independent phospholipase A 2

after 25 min, 3 h, and 24 h for paracetamol