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Together, these data reveal an enteric neuronal VIP-VPAC 2 ILC3 signaling pathway that is important for the dynamic control of IL-22 production in the gut and that increased protection of the epithelial barrier is linked to feeding, when the luminal content of the gut is greatest

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During autophagy, LC3-I undergoes ubiquitin-like conjugation and processing to be coupled with PE to form LC3-II, which then localizes on the inner and outer membranes of autophagosomes

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Additionally, after prolonged use, bacteria can resist AMPs (Pero et al