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doi: 10.1016/j.brainres.2007.09.019

Importantly, the genetic and pharmacological inhibition of MT-1G enhances the anticancer activity of sorafenib in vitro and tumor xenograft models (Sun et al., 2016a)

Recent studies highlight a potential therapeutic role for MR in cancer metabolism as many tumors exhibit a higher requirement for exogenous methionine, either due to an increased metabolic need for this amino acid in the maintenance of accelerated tumor growth or due to poor methionine recycling from homocysteine [202,203]

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[DOI] [PubMed] 853.Chifotides HT, Bose P, Verstovsek S

Hyperactivation of NF-B and suppression of peroxisome proliferator-activated receptor gamma (PPAR) pathways further reinforce this pro-ferroptotic phenotype