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ACS Appl Mater Interfaces 5:52395246

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Exogenous lipoate caused no substantial changes in GSSG/GSH levels but led to an increase in lipoyl-GMP, suggesting activation of lipoate consistent with use of the salvage pathway for protein lipoylation (Fig

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FASN uses acetyl-CoA as a substrate to synthesize saturated fatty acids (SFAs), whereas SCD1 can convert SFAs to MUFAs, thereby reducing the sensitivity of cells to ferroptosis [27,28,29]

GR depends on sufficient NADPH availability