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Figure 6 4.5.1 Iron dyshomeostasis in AD pathogenesis As early as the 1960s, cortical iron accumulation was identified as a pathological feature of AD (291)

Mol, Clin Environ Toxicol

The disruption of LLPS in the brain primarily is considered to stem from several factors: (1) mutations in proteins or expansions of low-complexity domains that favor pathological aggregation

Copper peptides prolong anagen while shortening telogen