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55 Proposed mechanisms include suppression of the JAK-STAT pathway, downregulation of receptor expression, impaired post-receptor signaling, decreased histone deacetylase 5 activity in the hypothalamus, elevated C-reactive protein, endoplasmic reticulum stress, and hypothalamic inflammation

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3 ), which results in the rupture of the outer mitochondrial membrane and the release of intermembrane proteins, including endonuclease G and apoptosis-inducing factor (AIF)

Nucleic Acids Res 50 , 72247234 (2022)

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