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Allergic reactions including angioneurotic edema have also been reported in postmarketing experience

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[3] [4] When approved, Apadaz received a labeling that highlighted all relevant aspects of the drug, including the lower abuse profile compared to traditional hydrocodone-acetaminophen

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(2016) [165], up-regulation of Grx1 in microglia cells results in proinflammatory signalling which ultimately leads to neuronal death in co-culture systems

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melanogaster mutants lacking PRX3 show few effects, supporting a functional redundancy of PRX5 and PRX3 54

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