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However, SMARCB1 deficiency (-SMARCB1) concordantly reduced the ATAC-seq, H3K27ac signals, and mRNA signals at the TSS site (Fig

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Transcriptomic analyses comparing nonpathogenic and pathogenic Th17 cells revealed that CRAC channels control mitochondrial but not aerobic glycolysis gene expression in pathogenic Th17 cells (79)

Moderate energy restriction-induced weight loss affects circulating IGF levels independent of dietary composition