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Presynaptic kainate receptors that enhance the release of GABA on CA1 hippocampal interneurons
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Although these defects have been shown to be correlated with lack of these nonessential genes, we dont hypothesize the impacts to this pathway are linked to genome instability based on the observed phenotypes to date
doi: 10.1016/j.bbamem.2008.10.009 149 GrabowskiG
Br J Diabetes Vasc Dis 12(4):189193
In the presence of sufficient reducing agents, such as GSH or NADPH, aconitase can be restored to its active form
