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doi: 10.3177/jnsv.64.1 18 LutsenkoS

(2010) 376, no

These findings suggest the presence of a compensatory pathway present in cells expressing both IR and IGF-1R through which loss of insulin generated signals triggers increased signaling via IGF-1/IGF-1R

Mazzei ME, Richeldi L, Collard HR (2015) Nintedanib in the treatment of idiopathic pulmonary fibrosis

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