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Assessing BWS and nonBWS liver tissue in this study provided insight into the functional consequences of 11p15 alterations between the normal and the overgrown liver that contribute to the BWS-specific cancer predisposition signatures, but the previous findings were limited, due to the bulk sequencing approach used

doi: 10.4172/2155-9562.1000209 215 KoprichJBJohnstonTHReyesMGSunXBrotchieJM

Voltage-dependent sodium channels such as SCN1A, SCN2A, SCN3A, SCN8A, SCN9A, and SCN1B, which encode Na v 1.1, Na v 1.2, Na v 1.3, Na v 1.6, Na v 1.7, and Na v 1, respectively, have had many different mutation sites reported for each gene, including both nonsense and missense mutations [26]

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The role of ESAT-6 in tuberculosis immunopathology

Such a strategy was also adopted by the authors of the previously cited various vitamin B12 desensitization protocols [9,10,11,12,13,14,15,16]