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doi: 10.1126/scisignal.2005375 [DOI] [PMC free article] [PubMed] [Google Scholar] 143

doi: 43

This chelation dramatically enhances copper bioavailability and facilitates cellular copper uptake through mechanisms that may involve cell-surface receptors and endocytic pathways

The new study extends this concept to skin inflammation and proposes that increased FABP5 expression funnels these redox-active lipids toward cellular membranes while suppressing the expression of GPX4, the lipid peroxidationdetoxifying enzyme and key ferroptosis suppressor, thereby amplifying lipid peroxidation and promoting inflammatory cell recruitment (Fig

Description GHK-Cu (Glycyl-L-histidyl-L-lysine copper complex) is a naturally occurring tripeptide-copper complex found in human plasma, saliva, and urine