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JS-K is reportedly activated by glutathione conjugation by glutathione transferase (GST), but the scope of activities among the numerous members of the GSTome is unknown

one-way ANOVA and Dunnetts post-hoc test for each timepoint

Additionally, the in vivo administration of Gln may drive NO production by delivering additional substrate (l-arginine) to NOS [15]

Conversely, overexpression of the mitochondrial biogenesis factor PGC1 enhances mitochondrial function and facilitates persistence of virus-specific and tumor-infiltrating cytotoxic T cells, suggesting a potential therapeutic target to augment T cell-mediated cancer immunotherapy [15,36]

Pak J Sci Ind Res B Biol Sci

M.YangC