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Even though oral treatments of the methyl donor SAM ameliorated ASD-associated deficits in valproic acid (VPA)-exposed mice by reversing epigenetic changes, there has been limited success thus far in the development of preventative agents similar to SAM for the clinical management of ASD in humans

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A 96-well microplate reader capable of reading optical density at 450 nm is required for use of this kit

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Therapeutic strategies developed and tested in the past Significant progress has been made by the neurofibromatosis research community, including the genetic cause of NF1, the cellular function of the neurofibromin protein, and the identification of cellular components that contribute to the tumours developed in NF1