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The neuroactive steroid pregnenolone sulfate stimulates trafficking of functional N-methyl D-aspartate receptors to the cell surface via a noncanonical, G protein, and Ca 2+ -dependent mechanism

34 ER stress-mediated Ca 2+ influx through the Ca 2+ channel ORAI calcium release-activated calcium modulator 1 (ORAI1) contributes to GSH depletion-induced ferroptosis in neuronal cells

19 The bioavailability of orally administered cysteine is believed to be the rate-limiting step for the synthesis of glutathione, 10 although the amino acids glutamate/glutamine are also integral to GSH synthesis

The injections themselves are self-administered not into a vein but an area of fatty tissue, such as the belly, thigh, or upper arm

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doi:10.1182/blood-2010-08-303859 589. Anbazhagan AN, Ge Y, Priyamvada S, Kumar A, Jayawardena D, Palani ARV, Husain N, Kulkarni N, Kapoor S, Kaur P, et al