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Further mechanistic insights reveal that glucose-starved cells with high expression of SLC7A11 leads to abnormal disulfide bonding within actin cytoskeleton proteins (ACPs), which results in the breakdown of the actin network, detachment of the plasma membrane, and ultimately disulfidptosis cell death (Liu D

Another pathway is that MSCs and their secretome, consisting of soluble factors and extracellular vesicles (EVs), can yield beneficial effects in kidney diseases through the paracrine release of over 1500 bioactive components, including functional peptides, proteins, mRNAs, microRNAs (miRs), and lipids [21, 84]

PMID: 18347346

doi: 10.1016/j.neuint.2017.04.016

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