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Moreover, VEGF preconditioning facilitates NSC remodeling and supports structural and functional resilience during aging [213]

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One is to inhibit processes or pathways that lead to the damage of cochlear cells

M1 and M2 subpopulations generally exhibit opposing functions: M1 macrophages release pro-inflammatory cytokines, while M2 macrophages display an anti-inflammatory phenotype involved in tissue repair and healing [25]