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Mechanisms of mtorc1 activation by rheb and inhibition by pras40

A scapegoat for accelerated neurodegeneration in Parkinsons disease

Because ob/ob mice administered LNP-anti-miR-30e-5p or LNP-Control did not significantly differ in lipid profile or body weight (Supplemental Figure 5D, 5E), the mechanism by which LNP-anti-miR-30e-5p reduced atherosclerosis would be independent of hyperlipidemia (Figure 7L-M)

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