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This regulatory function occurs via macrophage polarization in response to both internal and external environmental or pathological stimuli (Ying et al

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Erythrocytes, for example, generate a large amount of NADPH through the pentose phosphate pathway to use in the reduction of glutathione

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This suggests that loss of Nf1 does not induce muscle atrophy at a specific time point, but rather that NF1-associated muscle weakness is a continuous process of decreased hypertrophic growth beginning in childhood